![]() However, not all retinal tears occurring in eyes with lattice degeneration occur adjacent to a lattice lesion, suggesting that these eyes may be at a generalized increased risk of retinal tears. This traction may originate from a posterior vitreous detachment, and as such, the retinal tear is often on the posterior margin of the lattice lesion. Retinal tears are believed to stem from traction at the margins of lattice lesions, an area typified by tight vitreoretinal adhesion. Interestingly, these retinal detachments occur more frequently in young, myopic patients, an observation possibly explained by the progressive strengthening of the bond between the retina and retinal pigment epithelium at the border of the hole with time. And conversely, the risk of a retinal detachment developing in a patient with lattice degeneration associated with atrophic hole has been estimated to be less than 0.3%. Overall, a small percentage of retinal detachments are caused by lattice degeneration with atrophic holes (2.8% from one study). As this cuff typically remains stable over time, it is believed that the overlying liquid vitreous does not communicate with the greater vitreous body. These holes rarely progress to a clinical retinal detachment though may develop a small cuff of subretinal fluid stemming from the overlying liquefied vitreous. This occurs via two mechanisms: (1) atrophic retinal hole or (2) retinal tear.Ītrophic round holes occur within the substance of the lattice lesion and likely represent the end-stage of retinal thinning and subsequent dissolution of tissue. It is well documented that lattice degeneration increases the risk of retinal tears or subsequent retinal detachments. Lack of basement membrane over the surface of lattice lesions and replacement with glial cells.Tight vitreoretinal adhesion at the margins of the lesionĮlectron microscopic studies of lattice lesions demonstrated presence of the following features :.Vitreous liquefaction overlying the thinned retina. ![]() Postmortem histologic studies of lattice lesions have exhibited three invariable findings : ![]() The etiology of lattice degeneration is unknown, though theories include developmental anomalies of the internal limiting membrane, embryologic vascular anastomosis, localized retinal ischemia, or the contention that it is a primary vitreopathy leading to the formation of abnormal vitreous traction. Wagner Syndrome may also present with lattice degeneration. This feature is not believed to be congenital, but rather, develops during childhood and progresses throughout life.
0 Comments
Leave a Reply. |
AuthorWrite something about yourself. No need to be fancy, just an overview. ArchivesCategories |